AANEM Monograph: NEE Flashcards

(37 cards)

1
Q

Information provided by NEE

A

confirm dx. exclude mimics, identify unrecognized, localize, severity, pathophysiology, prognosticate

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2
Q

NEE technique

A

pull skin, insert needle (resting), withdraw to sub q then antigravity with insertion ID units morphology (duration/polyphasia/duraiton) recruitment (occ to interference pattern)

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3
Q

When should you quantitate

A

for mild/early processes

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4
Q

Coagulopathy avoid

A

diaphragm, TA, iliopsoas,fpl, pull skin avoid <30k plat

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5
Q

6 firing patterns

A

regular (<1% variation) - regular no change - regular with exponential change - irregular - semi rhythm (10% variation) - bursts

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6
Q

Origins: single fiber firing alone

A

endplate, fibs ( spike/psw), myotonic)

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7
Q

Origins: single fiber firing in groups

A

insertional - CRDS

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8
Q

Origin: Motor unit - individual spont

A

fasciculation - neuromyotonic - myoclonus - dystronia - stiffman

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9
Q

Origin: Motor unit - bursts spont

A

myokmic - synkinesis - termor - hemifacial spasm

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10
Q

Origin: voluntary

A

motor unit potentials

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11
Q

6 factors affecting Motor unit morphology morphology and synchrony

A

Reinnervation - collateral sprouting - loss of fibers - fiber splitting - atrophy - fiber regeneration

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12
Q

Recruitment frequency in limb and cranial

A

limb - 7-10 hz

cranial 16hz

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13
Q

Poor activation reasons

A

UMN, Pain, cooperation, strong muscle, two joint muscles (gastroc)

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14
Q

Goal rise time

A
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15
Q

When looking at 1-4 MUAPs note the

A

rise time < .6ms - duration (5-15ms) - polyphasia <5 - amplitude <20 - 200 microv - stability(MMAV)

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16
Q

increased insertional activity

A

> 200ms, seen in denervation and mytonic disorders

17
Q

fibrillation potentials reflect

A

muscle fiber diameter

18
Q

Main categories associated with fibrillation potentials

A

LMN - NMJ - Myopathies - muscle trauma

19
Q

disease associated with myotonic potentials

A

myopathy with or without clinical myotnia and neurogenic disorders

20
Q

CRDS usually indicated

A

chronic neurogenic, myopathic, normal in iliopsoas and biceps

21
Q

ALS: fascics without fibs =

A

not sufficient for dx

22
Q

situations associated with fascics

A

hyperthyroid, mestinon , normal in BFS, cramp fasciculation syndrome, neurogenic

23
Q

neurotonic discharge

A

interoperative nerve irritation

24
Q

2 situations for synkinesis

A

bell’s - arm-diaphragm

25
First change in a neurogenic lesion
reduced recruitment
26
4 situations that cause long duration MUAPs
increased fiber density, fiber number, loss of synchrony, collateral sprouting, reinnervation
27
diseases causing long duration MUAPs
chronic neurogenic, myopathies
28
diseases causing short duration
myopathy - nmj - early reinnervation or late stage atrophy - periodic paralysis
29
diseases causing unstable MUAPs
NMJ, neurogenic (reinnervation/progressive) - inflammatory myopathies
30
MUAPS in tremor fire in ___.
groups
31
Patterns: Normal recruitment with short duration polyphasic MUAPs
with variation - severe NMJs occ primary myopathies - | without variation - primary myopathies
32
Patterns: normal recruitment and normal muaps
normal and some metabolic myopathies
33
Patterns: reduced mixed durations
chronic myopathies - no variation in acute neurogenic but subacute there can be
34
Patterns: Reduced short duration polyphasic
without variation severe myopathy, end stage neurogenic disorder with variation early/ongoing reinnervation
35
Patterns: Reduced long duration polyphasic
chronic neurogenic or if variation then ongoing neurogenic lesion
36
Patterns: Reduced mixed durations
rapid neurogenic lesion
37
Patterns: Rapid recruitment
instability except for mild myopathies - long duration in chronic myopathies short duration in all other myopthies.