abnormal labour + delivery + ob emergencies part II Flashcards

1
Q

RF for breech

A
greater parity
multiples
hydramnios
oligohydramnios
hydrocephalus
anencephalus
prev hx
uterine anomalies
pelvic tumour
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2
Q

types of breech

A

frank - knees straigh

complete - knees bent

partial - footling/kneeling

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3
Q

risks /w breech birth

A
birth trauma + asyphxia (head entrapment)
preterm delivery or IUGR
cord prolapse
placenta previa
fetal anomalies
uterine anomalies/tumour
multiples
operative delivery
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4
Q

reason for head entrapment in breech delivery

A

less time for moulding

head biggest part

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5
Q

management of breech position

A
  • diagnose at 35-36wks
  • ECV after 36 wks
  • and/or planned CS
  • ## or trial of vag breech birth
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6
Q

risks of ECV

A
  • abruption
  • hemorrhage
  • ROM
  • labour
  • fetal bradycardia
  • cord accident
  • allo-immunization (give Rhogam)
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7
Q

selection criteria for vag delivery of breech

A
  • pre or early labour US to assess type of breech (CS if unavailable)

CI if:

  • cord presentation
  • fetal growth restriction
  • macrosomia
  • not frank or complete
  • inadequate maternal pelvis
  • fetal anomaly (eg hydrocephalus)
  • preg women refuses TOL

Can be offered if
- EFW 2500-4000g

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8
Q

breech labour management

A

check for abnormal pelvic contraction

good progress = room likely, CS if not

cont FHR

check for prolapse when rupture

induction not recommended, oxytocin can be used

active second stage near OR

paeds team at delivery

CS if delivery not imminent after 60min pushing

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9
Q

RF for shoulder dystocia

A
post-term
maternal obesity
fetal macrosomia
prev hx
operative vag delivery
prolonged labour
poorly controlled diabetes
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10
Q

risks of shoulder dystocia

A
  • PPH/uterine atony
  • maternal laceration
  • uterine rupture
  • birth injury
  • clavicle, humerus #
  • brachia plexus injury
  • hypoxic ischemic encephalopathy
  • death
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11
Q

maneuvers for shoulder dystocia

A
A - ask for help
L - lift legs (mcroberts)
A - anterior shoulder dysimpaction (suprapubic pressure + vag shoulder pressure)
R - rotate (woods corkscrew)
M - manual delivery of posterior arm
E - episiotomy (+ deliver arm)
R - roll onto all 4s

after this:
clavicle fracture
symphysiotomy
zavanelli maneuver

don’t

  • pull
  • pivot
  • panic
  • push on fundus
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12
Q

delivery options for future preg after dystocia

A

risks and benefits discussed /w patient

review notes

TOL or CS options

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13
Q

causes of poor oxygenation to fetus

A

maternal

  • anemia
  • carboxyhemaglobin
  • hypotension
  • regional anesthesia
  • position
  • vasculopathies (T1DM, SLE, HTN)
  • antiphospholipid antibody

utero-placental

  • hyperstim
  • abruption
  • infarction/oligohydramnios
  • chorioamnionitis

fetal

  • cord compression
  • oligo
  • cord prolapse, knots
  • fetal hemaglobinopathy
  • anemia (isoimmunization, fetomaternal bleed)
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14
Q

definition of fetal asyphxia

A

diagnosed at birth, must have all of:

  • apgar 0-3 for >5min
  • neonatal neuro sequelae
  • multi organ dysfunction in neonate
  • UA pH <7.0 and base deficit >16
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15
Q

pathophysiology and interpretation of acid/base

A
  • respiratory acidosis normal, CO2 accumulates with cord compression, goes away /w first breaths
  • metabolic acidosis is from hypoxia –> anaerobic metabolism and lactic acid, causes longer to develop + remit, potential for damage

Resp: increased PCO2 and normal base deficit

metabolic: N PCO2 and decreased base deficit
mixed: increased PCO2 and decreased base deficit

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16
Q

pathophysiology and interpretation of acid/base

A
  • respiratory acidosis normal, CO2 accumulates with cord compression, goes away /w first breaths
  • metabolic acidosis is from hypoxia –> anaerobic metabolism and lactic acid, causes longer to develop + remit, potential for damage

Resp: increased PCO2 and normal base deficit

metabolic: N PCO2 and decreased base deficit (bicarb low)
mixed: increased PCO2 and decreased base deficit

17
Q

causes of fetal bradycardia

A
  • maternal hypotension
  • drugs
  • fetal hypoxia (cord prolapse/compression, abruption, fetal hypotension, fetal hemorrhage)
  • vagal stimulation
  • fetal acidosis
  • fetal cardiac conduction or structural defect
18
Q

causes of fetal tachycardia

A

maternal

  • fever
  • infection
  • dehydration
  • hyperthyroidism
  • anxiety
  • drugs
  • anemia

fetal

  • infection
  • prolonged activity
  • chronic hypoxemia
  • cardiac abnormality
  • congenital anomalies
  • anemia
19
Q

variable decels causes

A
  • vagal stimulation (head compression)
  • cord compression
  • complicated: fetal acidemia
20
Q

early decels causes

A
  • vagal head compression
21
Q

late decels causes

A
  • altered blood flow to placenta
  • reduced mat O2 sat
  • uterine hypertonus
  • fetal acidemia
22
Q

decreased variability causes

A
  • acidemia if /w decels
  • maternal acidemia + analgesics can decrease variability
  • occurs /w tachycardia
23
Q

normal intrapartum tracing

A
  • baseline 110-160
  • variability 6-25 or <5 for <40min
  • decels: none or occasional uncomplicated variables or early decels
  • accels: spont present, at minimum present /w scalp stim
    action: can interrupt FHR for 30 min if stable and oxytocin stable

no evidence of fetal compromise

24
Q

atypical intrapartum tracing

A
  • bradycardia 100-110
  • tachy >160 for 30-80 min
  • rising baseline
  • variability <5 for 40-80min
  • decels: repetitive uncomplicated variables (3 in row), occasional (<2 in a row) complicated variables, intermittent lates, single prolonged decel 2-3min
  • accels: none /w scalp stim
    action: vigilant assessment. Likely physiologic response.
25
Q

abnormal intra-partum tracing

A
  • bradycardia <100bpm
  • tachycardia >160 for >80min
  • erratic baseline
  • variability <5 for >80min
  • > 25 for >10 min
  • sinusoidal
  • repetitive complicated variables:
  • > 60s below baseline
  • > 60s and <60bmp
  • loss of variability
  • biphasic
  • overshoots
  • slow return to baseline
  • baseline lowers
  • baseline tachy or brady
  • late decels > 50% of contractions
  • single prolonged decel >4min but <10min
  • accels absent usually
    action: scalp pH and/or delivery
26
Q

management of abnormal/atypical tracing

A
  • reposition
  • maternal vital
  • IV fluid bolus
  • stop oxytocin
  • O2 at 8-10 L/min
  • rule out fever, dehydration, drugs, prematurity
  • cont FHR
  • fetal scalp sample if appropriate
  • delivery if needed/persists
27
Q

fetal scalp sample intepretation

A

pH >7.25 normal

pH 7.21-7.24: cont, repeat in 30 mins or if change

pH <7.20 - deliver

for lactate:
<4.1
4.2-4.8
>4.8

28
Q

meconium significants

A

theories

  • distress, hypoxia
  • normal GI
  • vagal stim from transient cord compression

aspiration can occur if acidemia

RF: post-term + IUGR

29
Q

mech aspiration risks to baby

A
  • pulmonary hypertension
  • mechanical airway obstruction
  • chemical pneumonitis
30
Q

cord prolapse RF

A
  • malpresentation
  • prematurity
  • anomalies
  • multiples
  • polyhydramnios
  • premature ROM
  • AROM
  • placenta previa
  • high presenting part
  • ECV
31
Q

management of cord prolapse

A

1) assess viability: no cord pulsations, lethal anomaly, or too immature to survive –> deliver vaginally, CS if CI (eg transverse lie)
2) relieve cord compression, replace into vagina, elevate presenting part, adjust mom in trendelenburg
3) if fully dilated and low: assisted vag delivery, otherwise immediate CS