Exam 3 Flashcards

1
Q

normal intrapleural pressure is

A

negative compared to atmosphere

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2
Q

pleural disorders result from:

A
Change in amount of pleural fluid
Infections
trauma
congenital malformations
These disorders restrict lung expansion secondary to inflammation, fibrosis or other fluid in the pleural space
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3
Q

Pleurisy (what is it and causes)

A

Pleural inflammation
Definition- acute inflammation of parietal pleura
Causes: pneumonia, pulmonary infarction, rib injury, viral respiratory illness

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4
Q

Pleurisy symptoms

A

Sharp stabbing pain with any movement of the chest wall(especially inspiration), dyspnea, fever, abrupt onset of pain, unilateral, Crackles, pleural friction rub, Decreased breath sounds, area tender (pt can pin point location of pain)

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5
Q

Pleurisy Diagnosis and treatment

A

Medical treatment-relief of pain and treat underlying cause

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6
Q

Pleural Effusion (what is it)

A

Accumulation of fluid in the pleural space
normally, fluid seeps in from parietal pleura capillaries and is reabsorbed by visceral pleura capillaries and lymphatics- any condition that changes either of these can lead to pleural effusion
(increased hydrostatic, hyperalbumin, trauma, infection)

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7
Q

Pleural Effusion clinical manifestations

A

Depends on the size of the effusion
-less than 250ml may only be seen on CXR
-large effusion-lung expansion will be restricted and pt. may have dyspnea especially on exertion
dry non productive cough-due to bronchial irritation and mediastinal shift
-decreased tactile fremitus and breath sounds

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8
Q

Empyema

A

PUS IN THE PLEURAL CAVITY
must be drained and infection treated
may need surgical intervention if it becomes solidified and compresses the lung

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9
Q

Thoracentesis (what is it used for)

A

Used to remove fluid or air from the pleural space
used to decrease symptoms and diagnose source of fluid
note color and consistency of drainage
send specimen to the lab for: specific gravity, Glucose, Protein, pH, C&S, cytology

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10
Q

Pre-procedure thoracentesis

A

Informed consent, explain procedure
sit upright while leaning over a table
insertion of needle painful- medicate for pain
important to remain still during procedure- sudden movements can damage lung
takes ten to fifteen minutes to drain

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11
Q

Care during thoracentesis

A
Assist the doctor
provide emotional support to the patient
monitor the vital signs
observe for dyspnea, nausea, pain
encourage the patient to be still
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12
Q

Post-care thoracentesis

A

Turn to unaffected side for one hour for lung expansion
VS per institution policy, assess breath sounds
monitor for hemoptysis
record amount of fluid removed
CXR to assess lung reexpansion and presence of pneumo
assess for crepitus- mark the amount

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13
Q

Rib Fractures

A

One of the most common chest injuries
simple fracture-nondisplacing, little or no damage to the underlying tissues
treat with analgesics
symptom- pain at site of injury
CXR to confirm
Pt. Must cough and deep breathe
Monitor for 24-48 hours for lung contusion (ARDs)

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14
Q

Flail Chest (what is it)

A

Fracture of 2 or more ribs on the same side and possibly the sternum. Each rib is fractured in two or more places.
Can develop hemothorax if fractured rib tears pleura
chest wall is unstable
paradoxical chest movement with respiration-in with inspiration out with expiration

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15
Q

Flail Chest (signs and symptoms)

A

Pt can develop pulmonary edema, pneumonia, and atelectasis because fluids tend to increase and collect at injured site
altered chest movement
decreases patients ability to attain normal tidal volumes and cough effectively
hypoxemia and hypercapnia result
fear and fatigue due to pain with breathing

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16
Q

Flail Chest Diagnosis and treatment

A
  • Ineffective airway clearance, alteration in comfort, impaired gas exchange, ineffective breathing pattern
  • medical treatment depends on the severity of the case. Surgical stabilization and mechanical ventilation may be necessary
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17
Q

Pneumothorax (what is it, open/ closed)

A

Air in the pleural space- prohibits complete lung expansion
Closed pneumothorax- due to puncture or tear in internal respiratory structure (bronchiole, alveoli). Fractured rib can lead to this
Open pneumothorax- air enters pleural space directly through a hole in chest wall secondary to trauma

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18
Q

Pneumothorax symptoms (early)

A

Acute chest pain especially with chest movement, breathing, or coughing
apprehension (anxiety, restlessness)
Dyspnea, tachypnea, tachycardia, asymmetrical chest expansion
decreased or absent breath sounds on the affected side

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19
Q

Chest tube placement for pneumothorax

A

2nd intercoastal midclavicular line

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20
Q

Chest tube placement for hemothorax

A

5th/6th intercoastal mid-axillary line

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21
Q

Pneumothorax symptoms (late)

A

Distended neck veins, crepitus, decreased tactile fremitus, progressive cyanosis
tracheal deviation toward the unaffected side
pneumothorax is confirmed by CXR-chest tube must be placed

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22
Q

Open Pneumothorax

A
  • Opening in chest wall is big enough for air to move freely in and out of the chest cavity with ventilation
  • audible sucking noise
  • must emergently cover the wound
  • dressing of choice is sterile petrolatum gauze 4x4 and tape (not all 4 sides)
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23
Q

tension pneumothorax

A

Air enters the pleural space with each inspiration and becomes trapped (not expelled with expiration)
frequently associated with flail chest injuries, blunt traumatic injuries
if untreated collapses lung on unaffected side due to mediastinal shift
immediate intervention is required

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24
Q

Hemothorax

A

Blood in the pleural space
if small amount it may be reabsorbed (<300 cc)
if severe, 1400-2500cc, life threatening due to hypovolemia. Would also cause pressure on the unaffected lung.
Manifestations- tachycardia, hypotension, shock
treat with IV fluids(blood) and chest tube to suction

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25
Q

Chylothorax

A

Lipid-like fluid that accumulates in the pleural space
After cardiothoracic surgery
Treatment: Chest tube to drain
Decrease amount of fats and lipids pt receives

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26
Q

Thoracic Surgery

A

Wedge resection- pie like piece
segmental resection- larger section
lobectomy- lobe of lung (r-3, l-2)
pneumonectomy-only one that won’t need chest tube, postion on effected side (whole lung)
can be necessary because of lung cancer or possibly trauma

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27
Q

Pre-opertive Care pneumonectomy

A

Pulmonary function test will be performed
Alleviate anxiety
assess tidal volume, minute volume, vital capacity
Teach about post op expectations: presence of chest tubes, intubation and mechanical ventilation, oxygen therapy, available pain relief measures
Teach post op exercises, respiratory exercises, arm and shoulder exercises to maintain normal range of motion (shoulder is main problem), splinting

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28
Q

Post-op care pneumonectomy

A

Monitor airway- observe for respiratory failure
observe for signs of tension pneumothorax, crepitus especially if close to trachea
position for maximal expansion of lung tissue and gas exchange
Range of motion exercises to prevent shoulder
Increasing size of crepitus area
Monitor fluid status
pneumonectomy pts are more likely to get fluid overload due to IVFs and no CTs

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29
Q

Chest tubes (what for)

A

Required if:
sufficient air or fluid in the pleural space and ventilation is compromised
conditions that cause loss of negative intra-pleural pressure
PURPOSE:
remove air and fluid and restore normal negative pressure

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30
Q

Purpose of Drainage Systems

A

Provide for collection of drainage
provide one way system to prevent air or fluid from returning to the chest (water seal is the one way valve)
provide for control of suction to assist with removal of air and fluid

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31
Q

Reasons for a chest tube

A

Pneumothorax, Hemothorax, Tension Pneumothorax, Flail Chest, Pleural effusion, empyema, to prevent cardiac tamponade post open heart

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32
Q

Closed Pneumothorax

A

blunt trauma

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33
Q

Pneumothorax and hemothorax children

A

account for half of childhood intrathoracic injuries

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34
Q

clinical indications for chest tube

A
Physical Exam
-Decreased breath sounds or change in pitch
-Tachypnea
-Increased Respiratory Effort
-Sudden decrease in O2 saturations
Tension Pneumo
-agitation, hypotension from obstruction of venous return, severe hypoxemia, unilateral chest wall movement, tracheal deviation
Subcutaneous air (crepitus)
-Feels like Rice Krispies when palpated
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35
Q

key points for set up of drainage system

A

For set up:

  • Must use sterile water; CANNOT use sterile saline
  • Only fill Water Seal Chamber to the O or where the dotted 2cm line is (if over pt may have trouble breathing)
  • Only fill suction control chamber to order pressure (usually -20 cm H2O)
  • Adjust suction, after securing chest tube, so that gentle bubbling, NOT vigorous bubbling occurs in the suction chamber
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36
Q

Key points for monitoring a drainage system

A

Each shift, check the water levels in each chamber. Be sure to briefly stop suction to check suction chamber. Add water when levels are low. Only add water with suction stopped, then restart suction
If the water levels are overfilled, you can remove the water.
-Scrub the corresponding port on the back of the system
-Use a 20 gauge needle or smaller to access the port
Assess for air leak by looking for right to left bubbling in the water seal chamber
Ensure all connections are tight
Assess dressing site to ensure it is occlusive

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37
Q

Milking

A

Involves manipulations such as squeezing, twisting, or kneading to create bursts of suction within the tubing and chest tube lumen, increases thoracic pressure

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38
Q

Stripping

A

Done by compressing the chest tube with the thumb and forefinger against the chest wall to prevent dislodgement
Using the other hand, employ a pulling motion down the tubing away from the chest wall
Increases thoracic pressure

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39
Q

Patient safety with drainage system

A

Always secure Atrium Box to floor with tape or use hooks to secure to bed
Ensure tubing is NEVER kinked off; Monitor patient to avoid laying or positioning on tubing
Even if transporting on water seal, always have suction available
Have hemostats and gauze and/or vaseline gauze available in case of dislodgement
Always replace a system that has been tipped over

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40
Q

key points for chest tubes

A

Tape all connections
Maintain water at correct level
Keep drainage system below the level of the chest(2-3 ft.)
NO dependent loops
Do not clamp. Clamping can cause a tension pneumo.
Milking or stripping CTs is not recommended- increased negative pressure can be damaging to the lungs

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41
Q

Tidaling

A
Tidaling-water seal chamber fluctuates with respirations(fluid rises with inspiration and falls with expiration).  
DO NOT see tidaling when:
lung has fully reexpanded
when pts position kinks CT
when using suction.
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42
Q

Bubbling in the waterseal chamber:

A

-can be air moving out of the pleural space
Investigate continuous waterseal bubbling- can indicate a leak in the system- check chest insertion site, check tubing
-notify physician of sudden increase in bubbling of waterseal chamber
Waterseal chamber must have an airvent to provide escape route for air coming out of the pleural space

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43
Q

Chest tubes to suction

A

Continuous gentle bubbling in suction control chamber- lack of bubbling means not enough wall suction, if vigorous bubbling, water will evaporate
Usually 10-20cm of suction is used
The more fluid in the chamber, the more suction
Newer systems have float and dial

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44
Q

chest tube removal

A

Indications for readiness for CT removal:
-cessation of tidaling(when no suction)
-chest auscultation-adequate aeration on affected side
-CXR-no evidence of pneumo or fluid in pleural space
Usually a lung is re-expanded 2-3 days postop
Usually leave CTs in place 24 hours after all air and significant fluid drainage have stopped
sometimes temporairly clamped to see if pt can tolerate CT removal
Removal of CTs is very painful.
Pt must take a deep breath, hold his breath and bear down (to increase intrathoracic pressure) while CTs are being removed

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45
Q

After removal of chest tube

A

After removal, apply petrolatum gauze to insertion site followed by a 4x4 and tape
After removal the nurse must:
-observe dressing
-monitor for signs and symptoms of pneumothorax
-auscultate breath sounds per institution policy

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46
Q

Air leak?

A

check water seal chamber for bubbles

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47
Q

Drawing cultures from a chest tube

A

20g syringe, 30s CHG scrub, withdraw from tubing

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48
Q

Opioids

A

Morphine
Hydromorphone
Fentanyl
Reversal Agents: Narcan

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49
Q

benzodiazepines

A

Lorazepam (Ativan)

Midazolam (Versed)

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50
Q

Propofol

A

stops RR

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51
Q

Paralytics

A

Do not give without KO
vecuronium
rocuronium

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52
Q

Cholinergic blocking agents

A

Atropine
Ipatropium, (Atrovent)
Tiotropium (Spiriva)
increase HR

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53
Q

Adrenergic agents

A

Albuterol (Proventil)
Pirbuterol (Maxair)
Salmeterol (Serevent)
Levalbuterol (Xopenex)

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54
Q

Inhaled corticosteriods

A
Beclomethasone (Beclovent)
Budesonide (Pulmocort)
Fluticasone (Flonase, Flovent)
Triamcinolone (Azmacort)
Flunisolide (AeroBid)
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55
Q

Oral and parental corticosteriods

A

Hydrocortisone (Solu-cortef) (IV)
Dexamethasone (IV)
Methyprednisolone (Solu-medrol) (IV)
Prednisone (oral)

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56
Q

ETT

A

Endotracheal tube

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57
Q

WOB

A

work of breathing

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58
Q

VAP

A

ventilator associated pneumonia

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59
Q

Tidal volume

A

amount of air in normal breath

6-10 mL/Kg ideal body weight

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60
Q

I:E ratio

A

inspiratory to expiratory

normal is 1: 2( adult); 1: 1 (infant) -volume, flowrate, and rate control alter I:E ratio

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61
Q

PEEP

A

Peak End-Expiratory Pressure
used to improve oxygenation
keeps airway open

62
Q

Fraction of inspirated O2

A

FiO2= 0.4= 40% o2

63
Q

CPAP

A

is continuous positive airway pressure. It helps keep the alveoli open.
Can be used with mask or intubation or nasal prongs
Indicated for sleep apnea, COPD, pulmonary edema, heart failure, chronic or acute respiratory failure

64
Q

BiPAP

A

is bilevel positive airway pressure and is the same concept but different amount of pressure can be applied during inspiration and expiration

65
Q

Why intubate

A
Upper airway obstruction
Apnea
High Risk of Aspiration
Ineffective Airway Clearance
Respiratory Distress
66
Q

Intubation: Equipment

A

Ambu Bag, Mask, Oxygen, Suction setup, Oral Suction (Yankhauer), Suction Catheters, Endotracheal Tube (the right size) Adult: usually 7.0 – 9.0, Tape or Twill ties for securement, CO2 detector, Laryngoscope and Blade
Drugs
Will need to sedate and/or paralyze pt unless FULL ARREST

67
Q

Oral ETT

A
Cuffed or Uncuffed, Use larger size
Advantages:
-Decrease WOB because less airway resistance
-Easier to remove secretions
-Easier to do Bronchoscope
68
Q

Nasal ETT

A

Cuffed or Uncuffed, Use smaller size than oral, Used when head/neck manipulation is risky
DO NOT USE when: Facial fractures, Fracture at base of skull, Post-op cranial surgeries
Issues:
-Increased WOB due to smaller tube size
Some studies:
-Increased sinus infection risk
-Increased VAP risk

69
Q

Intubation Procedure

A

Provider places ETT
Confirm tube position:By auscultation of the chest, Bilateral chest rise, Tube location at teeth, CO2 detector – (esophageal detection device), CXR also used to confirm location of ET

70
Q

Intubation risks

A

Difficult to place (especially nasally)
Teeth may be chipped or broken
Increased salivation and increased difficulty swallowing
Obstruction because of biting
Decrease good oral care because of difficulty to use toothbrush
If nasal, then may kink easy

71
Q

Monitor for ETT placement

A

Monitor for placement every 2-4 hours
Confirm exit mark on tube remains constant while at rest AND while repositioning pt
Pt Assessment: Symmetrical chest rise and movement, Auscultate bilateral breath sounds

If dislodged, pt at risk for: Minimal or NO oxygen, No tidal volume (TV) delivered to pt OR one lung gets entire TV, Risk for pneumothorax

72
Q

Maintain proper cuff inflation ETT

A

Normal cuff pressure is 20-25 mmHg
Measure and record immediately after intubation and tube placement confirmation AND every 8 hours

Improperly inflated cuff leads to: Too much air leak, Too much pressure on trachea

73
Q

Indications for suctioning ETT

A
Visible secretions
Sudden onset of resp distress
Suspected aspiration
Increased peak airway pressures
Auscultation of adventitious breath sounds
Increased resp rate and/or coughing
Sudden or gradual decrease in PaO2
74
Q

complications of intubation

A
Unplanned Extubation
		*Signs of unplanned extubation:
		    -patient speaking
		   -low pressure alarm on ventilator
		   -decreased or absent breath sounds
		   -respiratory distress
		   -gastric distention
Aspiration
75
Q

Goals of Vent

A

Decrease work of breathing
Increase alveolar ventilation
Maintain ABG values within normal range
Improve distribution of inspired oxygen

76
Q

PCO

A

35-45

77
Q

SaO2

A

95-100

78
Q

PO2

A

80-100

79
Q

Volume modes

A

Volume is constant and pressure will vary with patient’s lung compliance. TV= mL per breath varies w/ pressure
Examples:
Assist-Control Mode (AC) (also known as Assisted Mandatory Ventilation (AMV)
Intermittent Mandatory Ventilation (IMV)
Synchronized Intermittent Mandatory Ventilation (SIMV)

80
Q

Pressure Modes

A

Pressure is constant and volume will vary with patient’s lung compliance.
Examples:
Pressure Support Ventilation (PSV)

81
Q

Volume mode- control mode

A

Delivers pre-set volumes at a pre-set rate and a pre-set flow rate.
The patient CANNOT generate spontaneous breaths, volumes, or flow rates in this mode.

82
Q

Volume mode- assist control

A

Delivers pre-set volumes at a pre-set rate and a pre-set flow rate.
The patient CANNOT generate spontaneous volumes, or flow rates in this mode.
Patient can initiate a breath but will be given the set volume of air
Each patient generated respiratory effort over and above the set rate are delivered at the set volume and flow rate.

83
Q

Volume mode- Synchronized Intermittent Mandatory Ventilation (SIMV)

A

Delivers a pre-set number of breaths at a
set volume and flow rate.
Allows the patient to generate
spontaneous breaths, volumes, and flow
rates between the set breaths.
Detects a patient’s spontaneous breath
attempt and doesn’t initiate a ventilatory
breath
alarm for low minute volume

84
Q

Pressure support

A

Extra support is provided with each inspiration
Pt must be spontaneously breathing
Preset- Inspiratory pressure, PEEP, Sensitivity
Patient determines: Own tidal volume, Own rate, Inspiratory time

85
Q

POSITIVE END EXPIRATORY PRESSURE (PEEP):

A

This is NOT a specific mode, but is rather an adjunct to any of the vent modes.
PEEP is the amount of pressure remaining in the lung at the END of the expiratory phase.
Utilized to keep otherwise collapsing lung units open while hopefully also improving oxygenation.

86
Q

CPAP Mode

A

This IS a mode and simply means that a pre-set pressure is present in the circuit and lungs throughout both the inspiratory and expiratory phases of the breath.
CPAP serves to keep alveoli from collapsing, resulting in better oxygenation and less WOB.
The CPAP mode is very commonly used as a mode to evaluate the patients readiness for extubation.

87
Q

Cardiac issues vent

A
Decreased venous return to the heart
Decreased left ventricular end-diastolic volume-Preload
Decreased Cardiac Output
Hypotension
can vegal
88
Q

Pulmonary issues vent

A
Barotrauma- increased pressure
Trauma from Overinflation
Alveolar Hypoventilation
Alveolar Hyperventilation
Ventilator-Associated Pneumonia
89
Q

Electrolyte issues vent

A

Fluid Retention- Especially with increased PEEP
Decreased urinary output
Increased Sodium Retention
Perhaps all from decreased cardiac output = decreased renal perfusion
Also, with stress response = release of ADH and cortisol which leads to sodium and water retention

90
Q

Neuro issues vent

A

Venous drainage from the head- JVD
With head injury…impaired cerebral blood flow
-Increased intracranial pressure because of impaired venous return and increased cerebral volume

91
Q

GI issues vent

A

Stress ulcers, GI bleeding, Ischemia of gastric and intestinal mucosa, Gastric and bowel dilation- From increased air swallowing, Decreased Peristalsis- From immobility, sedation, pain meds, stress and decreased cardiac output

92
Q

Musculoskeletal issues vent

A

Loss of muscle strength- Related to: sedatives, paralytics, lack of nutrition
Foot drop
Pressure Ulcers
Immobility

93
Q

Psychosocial issues vent

A

Physical and Emotional stress
Eat, speak, move or breath = NOT normal if ventilated
Feeling of being UNSAFE; If needs are met, patient will feel safe
4 needs:
Need to KNOW, Need to have CONTROL, Need to HOPE, Need to TRUST

94
Q

High Pressure Alarms

A

Secretions, coughing, gagging, patient fighting ventilator, water in tubing
Decreased compliance issues: Pulmonary edema, Pneumothorax
Increased resistance: bronchospasm, kinked tubing

95
Q

Low Pressure Limit

A

Ventilator disconnected!
Patient extubates self-partially or totally
Patient trying to speak
Cuff Leak

96
Q

Apnea

A

Oversedation, change in patient condition, total or partial extubation
High Volume
Pain, anxiety, change in patient condition
Low Volume
Cuff leaking, pt becomes disconnected from circuit (or loose connections), change in patient’s effort

97
Q

phases of weaning

A
  1. Preweaning
    * Consider respiratory and nonrespiratory factors
    * SBT (Spontaneous Breathing Trial)
  2. Weaning
    * Reduce settings gradually and assess how patient tolerates
  3. Extubation (Outcome)
98
Q

Criteria for extubation

A

TV of at least 5 ml/kg
MIP (maximum inspiratory pressure) of at least -20 cm H20. The more negative the number the more strength the pt has to pull in a breath.
Vital capacity (VC) of 10-15 ml/kg
Minute volume (MV) of at least 5 liters (TV X RR)
RR within normal limits WNL), not labored, not shallow, no use of accessory muscles
LOC WNL
O2 sat >95%
Heart rhythm WNL
They should be calm and able to follow verbal commands.
They should have the strength to lift and hold their arms up as well as lift their head off the pillow

99
Q

Patients who have a threat to__________ should be treated first

A

life, vision, or limb

100
Q

Uses 4 factors to determine Triage level

A

Stability of Vital Functions (ABCs)
Life threat or Organ Threat
How soon the pt should be seen
Expected resource intensity

101
Q

five level emergency index

A
1-threat to life
2-High Risk Situation, Confused, severe pain &amp; dangerous VS
3- Stable VS and need many resourse 
4-one resource needed
5-no resources needed
102
Q

primary survey-

A

Trauma care begins with the primary survey- rapid assessment of the patient’s ABCs with the addition of disability (what can kill the patient now)

103
Q

Followed by secondary survey

A

complete head-to-toe assessment to identify other serious injuries that could kill or disable the patient later

104
Q

Airway

A

Cervical Spine Stabilization &/or Immobilization (Cervical Collar, Cervical immobilization device (CID) – head blocks)

105
Q

Breathing

A

medical problems cause alterations in breathing—-fractured ribs, pneumothorax, penetrating injury, allergic reactions, pulmonary emboli, and asthma attacks
signs & symptoms of alterations in breathing
-Dyspnea, Paradoxical or Asymmetric chest wall movement, Decreased or absent breath sounds, Visible wound to chest wall, Cyanosis, Tachycardia and/or Hypotension

106
Q

Circulation

A

Two large-bore (14-16 gauge) IV catheters should be inserted & fluid resuscitation initiated using lactated Ringer’s solution or normal saline
Direct pressure with a sterile dressing should be applied to bleeding sites

107
Q

Disability

A

A brief neurologic exam completes the primary survey
The degree of disability is measured by the pt’s level of consciousness
Determining the pt’s response to verbal &/or painful stimuli is one approach to assessing level of consciousness
Pupils are assessed for size, shape, equality, and response to light
AVPU

108
Q

AVPU

A
A = Awake
V = Responsive to Voice
P = Responsive to Pain
U= Unresponsive
109
Q

Exposure/enviromental control

A

Secondary
All trauma patients should have their clothes removed
Once the patient is exposed, it is important to limit heat loss and prevent hypothermia by using warming blankets, overhead warmers, and warmed IV fluids

110
Q

History

A
Use AMPLE:
Allergies
Medication history
Past Health History
Last Meal
Events/Environment
provides clues to the cause of the crisis and suggests specific assessment and intervention needs
111
Q

Raccoon eyes

A

closed head injury, happens 2-3 days later, basal skull fracture

112
Q

Battle sign

A

bruising behind the ear

113
Q

Drainage

A

Do not block or clear drainage from ear or nose

Check ear for blood or cerebrospinal fluid

114
Q

Blunt Injury

A

Mechanisms of blunt injury include motor vehicle crashes (MVCs), falls, assaults, contact sports
Multiple injuries are common with blunt trauma & extent of injury is less obvious than penetrating injuries and the diagnosis can be more difficult.

115
Q

Penetrating injury

A

Refers to an injury produced by foreign objects penetrating the tissue
-stabbings, firearms
impalement- injuries that penetrate the skin and result in damage to internal structures
Damage is created along the path of penetration
Can be misleading because the condition of the outside of the wound does not determine the extent of internal injury

116
Q

Cullen’s Sign

A

Purplish discoloration of the flanks or umbilicus- indicative of blood in the abdominal wall

117
Q

turners sign

A

ecchymosis on flank- may indicate retroperitoneal bleeding or pancreatic injury

118
Q

Priapism

A

errection that doesnt go away

119
Q

FAST

A

Focused abdominal sonography for trauma- used to see if fluid is present
Cannot rule out Retroperitoneal bleed

120
Q

Internal hemmorage

A

If a patient shows no signs of external bleeding but exhibits tachycardia, fall in B/P, apprehension, cool and moist skin, or delayed capillary refill, internal hemorrhage is suspected
PRBCs are administered
Patient is prepared for more definitive treatment (surgery)
Maintained in supine position until hemodynamic parameters improve

121
Q

Heat exhaustion

A

Assessment Findings: Fatigue, Light-headedness, N/V, diarrhea, Feelings of impending doom, Tachypnea, Hypotension, Tachycardia, Elevated body temperature, Mild confusion, Ashen color, Diaphoresis
Treatment: place patient in cool area, oral fluid and electrolyte replacement or normal saline solution IV

122
Q

Heat stroke

A

Increased sweating, vasodilation, and increased respiratory rate deplete fluids and electrolytes.
Eventually, sweat glands stop functioning, so core temperature increases rapidly.
Assessment Findings: core temperature above 104° F, altered mentation, absence of perspiration, circulatory collapse, skin is hot, dry, and ashen.
Treatment:
Reducing the core temp rapidly (until 102)
Administration of 100% oxygen
Fluid & electrolyte imbalances are corrected

123
Q

Frostbite treatment

A

Controlled yet rapid rewarming is instituted- painful.
Part is elevated to help control swelling.
Dehydration, hyperkalemia, and hypovolemia are corrected.
After rewarming, hourly active motion of the affected digits is encouraged to promote maximal function
Rewarming- the extremity is placed in a 37 to 40 degree (98.6-104) circulating bath for 30 to 40 minute spans
Avoid tobacco, alcohol, and caffeine because of their vasoconstrictive effects

124
Q

degrees of Hypothermia

A

mild hypothermia 90°- 95°F
moderate hypothermia 87°- 90°F
profound hypothermia less than 87°F

125
Q

Hypothermia

A

Assessment Findings
-Shivering (diminished or absent at temperatures less than 92°F), Hypoventilation, Hypotension, Altered mental status, Areflexia, Pale, cyanotic skin, Blue, white, or frozen extremities, Arrhythmias, Fixed, dilated pupils
Treatment:
-Rewarm patient, Continuous ECG monitoring because cold-induced myocardial irritability lead to ventricular fibrillation

126
Q

Carbonmonoxide poisoning

A

will need blood gas to assess.

127
Q

Biological agents of terrorism

A
Anthrax, Plaque, Tularemia
-Can be treated with antibiotics
Smallpox
-Prevented with vaccination
Botulism
-Treated with antitoxins
Hemorrhagic fever
-No current treatment
128
Q

Chemical agents of terrorism

A

Sarin nerve gas – paralyzes respiratory muscles
-Atropine, Pralidoximine chloride
Phosgene gas – causes respiratory distress and pulmonary edema
Mustard gas – causes skin burns and blisters

129
Q

Radiologic or nuclear agents of terrorism

A

RDD – radiologic dispersal devices (RDD) (dirty bombs)
-Mix of explosive and radiologic material
-Most damage from explosion, but should take measures such as covering nose and mouth and showering to limit exposure
Ionizing Radiation
-Damage to nuclear reactors or nuclear bomb
-Initiate decontamination procedure.
-Can develop acute radiation syndrome (ARS)

130
Q

Acute Respiratory Distress Syndrome (ARDS)

A

Sudden progressive form of acute respiratory failure
Alveolar capillary membrane becomes damaged and more permeable to intravascular fluid.
Alveoli fill with fluid.
Worse ALI

131
Q

ARDS results in

A

Severe dyspnea
Hypoxia- pO2 remains low despite increasing FIO2 (fraction of inspired oxygen)
Decreased lung compliance
Diffuse pulmonary infiltrates

50% mortality rate

132
Q

Etiology of ARDS

A

Develops from a variety of lung injuries
Most common cause is sepsis.
Direct lung injury: aspiration of gastric contents, pneumonia
Indirect lung injury: massive trauma, sepsis
Exact cause for damage to alveolar-capillary membrane not known
Pathophysiologic changes of ARDS thought to be due to stimulation of inflammatory and immune systems

133
Q

Neutrophils in ARDS

A

Neutrophils are attracted and release mediators, producing changes in lungs.

  • ↑ pulmonary capillary membrane permeability
  • Destruction of elastin and collagen
  • Formation of pulmonary micro emboli
  • Pulmonary artery vasoconstriction
134
Q

3 phases of ARDS

A

Injury or Exudative Phase
Restorative or Proliferative Phase
Fibrotic or Chronic Late Phase

135
Q

Injury or Exudative Phase

A

1 to 7 days after direct lung injury
-Damage to vascular endothelium, ↑ capillary permeability
-Engorgement of peribronchial and perivascular interstitial space- interstitial edema
-alveoli fill with fluid, and blood passing through cannot be oxygenated.
-Surfactant dysfunction → Atelectasis
-decreased gas exchange and decreased lung compliance
-V/Q mismatch- refractory hypoxemia.
Hypoxemia unresponsive to increasing O2 concentrations

136
Q

Restorative or Proliferative Phase

A

1 to 2 weeks after initial lung injury
Influx or neutrophils, monocytes, and lymphocytes
Fibroblast proliferation
Lung becomes dense and fibrous
Lung compliance continues to ↓ due to interstitial fibrosis
Hypoxemia worsens.
Thickened alveolar membrane
Diffusion limitation and shunting
If reparative phase persists, widespread fibrosis results.
If phase is halted, lesions resolve

137
Q

Injury or Exudate Phase assessment

A

↑ work of breathing
↑ respiratory rate
↓ tidal volume (stiff lungs)
Produces respiratory alkalosis from increase in CO2 removal (due to the increased RR)
Eventually have ↓ CO and ↓ tissue perfusion

138
Q

Fibrotic/Chronic or late phase

A

2 to 3 weeks after initial lung injury
Lung is completely remodeled by collagenous and fibrous tissues.
Results in
↓ lung compliance
↓ area for gas exchange with resultant hypoxemia
Pulmonary hypertension
Results from pulmonary vascular destruction and fibrosis

139
Q

ARDS early clinical manifestations

A

Dyspnea, tachypnea, cough, restlessness
Chest auscultation may be normal or may reveal fine, scattered crackles.
ABGs- Mild hypoxemia and respiratory alkalosis caused by hyperventilation
Chest x-ray may be normal or may show minimal scattered interstitial infiltrates.
-Edema may not show on CXR until 30% increase in lung fluid content

140
Q

ARDS late clinical manifestations

A

Symptoms worsen with progression of fluid accumulation and decreased lung compliance.
Pulmonary function tests:
-decreased compliance and lung volume.
Evident discomfort and increased work of breathing
-Tachypnea, suprasternal and intercostal retractions
Tachycardia, diaphoresis, changes in sensorium with decreased mentation, cyanosis, and pallor
Hypoxemia and a PaO2/FIO2 ratio <200 despite increased FIO2

141
Q

chest xray with ARDS

A

whiteout or white lung because of consolidation and widespread infiltrates throughout lungs

142
Q

Assessment of ARDS

A
Shallow breathing with increased respiratory rate
Tachycardia
Abnormal breath sounds
Somnolence, confusion, delirium
Changes in pH, PaCO2, PaO2, SaO2
Decreased tidal volume, FVC
Abnormal x-ray
Normal pulmonary artery wedge pressure (noncardiogenic pulmonary edema)
143
Q

Oxygen therapy with ARDS

A

Give lowest concentration that results in PaO2 60 mm Hg or greater to prevent O2 toxicity
Risk for O2 toxicity increases when FIO2 exceeds 60% for longer than 48 hours.
Patients will commonly need intubation with mechanical ventilation because PaO2 cannot be maintained at acceptable levels.

144
Q

Mechanical vent with ARDS

A

PEEP at 5 cm H2O
-Opens collapsed alveoli
Higher levels of PEEP are often needed to maintain PaO2 at 60 mm Hg or greater.
High levels of PEEP can compromise venous return.
-↓ preload, CO, and BP
High levels of PEEP can cause barotrauma
HYPOXIA is the issue and PEEP helps this

145
Q

Positioning with ARDS

A

Turn from supine to prone position.
Fluid pools in dependent regions of lung.
Proning typically reserved for refractory hypoxemia not responding to other therapies
Plan for immediate repositioning for cardiopulmonary resuscitation.
Continuous lateral rotation therapy (side to side turning via a rotation bed)

146
Q

Conplications of ARDS

A

Infection
Related to Mechanical Ventilation
-Ventilator-associated pneumonia
-Barotrauma (rupture of alveoli due to increased pressure)
-Prevent barotrauma with higher rates and lower TV
-High risk for stress ulcers
-Renal failure (decreased perfusion due to decreased CO)

147
Q

Ventilator Bundle Protocol

A
Elevation of the HOB 30-45 degrees
Daily “sedation holidays” and assessment of readiness for extubation
PUD prophylaxis
DVT prophylaxis
Oral care chlorhexidine solution
148
Q

Cor Pulmonale

A

Hypertrophy of the RIGHT Ventricle
-Secondary to diseases of lung, thorax, pulmonary circulation
May be present with or without cardiac failure
Results from pulmonary hypertension
50% of US cases are from COPD (Late manifestation)
-INCREASED pulmonary vascular resistance

149
Q

Cor pulmonale signs and symptoms

A
MOST COMMON:  Dyspnea on exertion
Tachypnea, cough
Lethargy, fatigue
Lung sounds:Normal or crackles in bases
Right-Sided Heart Failure S&amp;S:
JVD, Hepatomegaly, Right upper quadrant tenderness, Peripheral edema, Weight gain, Full, bounding pulse
150
Q

Diagnostics for Cor polmonale

A

H and P, ABGs, SpO2, Electrolytes, BNP, CXR, ECG, CT, MRI, Cardiac catheterization