Gait apraxia Flashcards

1
Q

Do we learn gait?

A

no it is instinctual, stepping movements are early in development

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2
Q

What patterns of gait are present at birth?

A

stepping patterns

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3
Q

What are operational synergies?

A

they are contained in the spinal cord in the form of central pattern generators (CPGs)

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4
Q

What can we do consciously to the organized synergies?

A

we can modify them based upon environmental demands

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5
Q

What structures are involved in gait?

A

integrated control of cortical areas, cerebellum, basal ganglia, and spinal cord

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6
Q

how do abnormalities of gait occur?

A

with dysfunction of a variety of nervous system structures

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7
Q

What is ataxic gait?

A

-wide base of support with irregular/erratic weight shifts and velocity

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8
Q

What structure is the origin of ataxic gait?

A

cerebellum

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9
Q

What is a tabetic gait? (aka sensory ataxia)

A

-wide base of support, high stepping (steppage), foot drop, irregular/erratic cadence, ataxia

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10
Q

What is tabetic gait due to?

A

peripheral nerve damage

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11
Q

What does the term tabetic gait come from?

A

tabies dorsalis - syphilitic cell death of DRG cells but may be due to other conditions

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12
Q

What is a hemiplegic (spastic gait)?

A

-slow, stiff leg, circumduction, foot drop, flexed arm posture with no swing

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13
Q

What is a hemiplegic gait caused by?

A

cerebral stroke

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14
Q

What is diplegic gait?

A

often faster, ataxic, stiff leg, circumducted, adducted, hip and knees flexed, plantar flexion, foot drop, flexed arm posture with no swing.

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15
Q

What pathology is common to have diplegic gait?

A

cerebral palsy

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16
Q

What is parkinsonian gait?

A
  • slow, stiff, shuffling gait, no arm swing, but can be a quick, short stepping
17
Q

What is a dyskinetic gait?

A

-rapid, fragmented movement intrusions, ataxia, dance like movement

18
Q

What is dyskinetic gait often caused by?

A

basal nuclei damage (caudate or subthalamic nucleus)

19
Q

What is apraxia?

A

the lack of motor plan to perform voluntary movement (inability to perform a task)

20
Q

What are the four forms of apraxia?

A

ideational
ideomotor
kinetic
oral

21
Q

What is ideational apraxia?

A

the inability to organize single actions into a sequence for intended purpose as in the loss of knowledge of the movement

22
Q

What is ideational apraxia caused by?

A

cortical in origin, no specific area, but parieto occipital area is very important

23
Q

What is ideomotor apraxia>

A

the inability to translate the idea of the action into an appropriate motor program

24
Q

ideomotor apraxia is lacking what?

A

proper sequencing of movement

25
Q

what is ideomotor apraxia most associated with?

A

damage to the supramarginal gyrus or superior parietal lobule

26
Q

What is kinetic apraxia?

A

form of clumsiness, loss of hand and finger dexterity, not due to paresis, ataxia or sensory loss.

27
Q

What is kinetic apraxia typically caused by?

A

damage to the pre motor cortex

28
Q

What is oral apraxia?

A

the inability to execute facial movements on command and is due to damage of the inferior frontal gyrus contiguous with broca’s area

29
Q

Why do oral apraxia and motor aphasia often coexist?

A

because of the anatomical proximity of the distributed neural networks subserving praxis and language exists in the left, dominant hemisphere