A. Amount of drainage since system was connected to patient
B. Level of resistance to drainage of pleural cavity
C. Level of underwater seal applied to pleural cavity
D. Maximum pressure ?in/against pleural cavity on expiration
E. Maximum suction that can be applied to pleural cavity
?
A. Atonic pupil B. Unilateral blindness in blocked eye C. Contralateral blindness D. Diplopia- past papers remembered this as dysphagia E. Nystagmus
C. Contralateral blindness
Caused by back-spread of LA to the optic chiasm
Drowsiness/Vomiting/Convulsions/Respiratory Depression/Arrest
http://bja.oxfordjournals.org/content/75/1/93.full.pdf
A. Accept that a small amount of bleeding may occur with a tourniquet
B. Reinflate at a higher pressure
C. Check coags
D. Take tourniquet down, rexanguinate and reinflate
E. Something else
A. Accept a small amount of bleeding
Aortic dissection
A. Avoiding daylight B. not sleeping during day C short naps during shift D use of caffeine or stimulants E. using benzodiazepines for sleep during the day
C
CEACCP - fatigue and the anaesthetist (2013)
6.Patient with Acute Intermittent Porphyria presents to hospital with abdominal pain and requires a general anaesthetic. Which drug for PONV would you avoid?
A. Metoclopramide B. Prochlorperazine C. Tropisetron D. Ondansetron E. Droperidol
A. Metoclopramide
Oxford handbook
A. adrenaline B. dobutamine C. ephedrine D. metaraminol E. phenylephrine
E. Phenylephrine
Moclobemide- reversible MAOI
Most dangerous - indirect sympathomimetics (Ephedrine/Metaraminol/Amphetamine/Cocaine)
Direct sympathomimetics - exaggerated effect
Serotonin Syndrome - Pethidine/Tramadol
Pancuronium - releases stored NA
Oxford Handbook
A. endobronchial intubation B. endotracheal cuff leak C. gas sampling line leak D. obstructive airways disease E. spontaneous ventilatory effort
C. Gas sampling line leak
A. T2 B. T4 C. T6 D. T8 E. T10
B. T4
A. Cease dabigatran 7 days prior
B. Cease dabigatran 3 days prior
C. Cease dabigatran 3 days prior and give bridging anticoagulation
D. Cease dabigatran 24 hours prior and measure INR on day of surgery
E. Continue dabigatran and withhold on day of surgery
B. Cease dabigatran 3 days, prior. No bridging required (low risk for thromboembolism).
Darbigatran - factor Xa inhibitor
At least 48 hours for spinal
MIMS
A. CXR B. CT chest C. MRI chest D. PET scan E. TOE
CT or MRI to determine presence/extent of airway compression. ?MRI better for 15 year-old to minimise radiation.
If she could tolerate it, would do this procedure under LA (if any concerns about airway compression or SVC syndrome).
A. GA B. Head compression C. Uteroplacental insufficiency D. Acute asphyxia E. Umbilical cord compression.
C. Uteroplacental insufficiency
Late decelerations begin at peak of uterine contraction and recover when the contraction ends.
Caused by:
Maternal Hypotension
Pre-Eclampsia
Uterine Hyperstimulation
Head compression- Early deceleration
Umbilical cord compression- Variable deceleration
http: //geekymedics.com/2011/05/29/how-to-read-a-ctg/
http: //ceaccp.oxfordjournals.org/content/3/2/38.full.pdf+html
A. 3 B. 4 C. 8 D. 25 E. 33
D. 25
1/ARR
1/Probability (with intervention)-Probablity (Control)
1/0.12-0.08=1/0.04 = 25
A. vecuronium B. cisatracurium C. pancuronium D. atracurium E. suxamethonium
B Cisatracurium
60-70% of all anaphylaxis
Anaphylaxis to suxamethonium - 60% to all others NMBD
Benzylisoquinolonium
Less potential for histamine release
Cisatracurium less histamine release
Peck + Hill
…If it’s elective, the safest thing to do is to defer the case until she has had allergy testing.
A. Gingko B. Garlic C. Ginger D. Fish Oil E. Echinacea
E. Echinacea
A. UTI B. PE C. Delirium D. AMI E. Pneumonia
C. Delirium
Anaesthesia UK - tutorial of the week: neck of femur fracture; perioperative management (2013)
A. Adrenaline B. Dobutamine C. Levosimenden D. Milrinone E. Vasopressin
E. Vasopressin
CEACCP - carcinoid: the disease and its implications for anaesthesia (2011):
A significant proportion of the surgery related to carcinoid will be for the removal of metastases by hepatic resection. Here, the need to try to maintain a relatively low CVP, during clamping of the hepatic artery and portal vein to avoid backflow into the liver and venous bleeding, will further exacerbate the risk of hypotension. The response to inotropic and vasopressor agents is unpredictable and, in general, drugs such as norepinephrine and epinephrine can be hazardous in carcinoid patients. Norepinephrine has been shown to activate kallikrein in the tumour and can even lead to the syn- thesis and release of bradykinin resulting paradoxically in further vasodilatation and worsening hypotension, although exaggerated hypertensive responses may be seen. Indeed, any pharmacological stimulation of the autonomic nervous system has the potential to provoke further problems with vasoactive hormone release. In practice, cautious administration of small doses of phenylephrine has been found helpful in some patients.
Vasoactive hormone release intra-operatively is best treated with intravenous boluses of 20 – 50 mg of octreotide, titrated to haemodynamic response. Vasopressin as an alternative vasocon- strictor that may be useful if prolonged vasoconstriction is required; however, the evidence base is small.
It must be borne in mind that concomitant fluid losses, especially bleeding, may be responsible for intra-operative instabil- ity rather than hormone excess and that fluid resuscitation may be the answer rather than further octreotide therapy. Monitoring of fluid losses, especially bleeding, is very important in these patients.
A. Instigate low dose beta blockade
B. Defer, and refer to a cardiologist
C. Perform a transoesophageal echo to get a better look at the valve
D. Proceed to surgery with no further investigation
E. Perform a dobutamine stress echo
D. Crack on
CEACCP - anaesthetic management of patients with hip fractures: an update (2013):
No study has so far shown that ‘preoptimization’ improves outcome among hip fracture patients, whereas two large meta-analyses of 275,000 patients have shown that delay to surgery beyond 48 h is associated with increased 30 day post- operative mortality, complications, and length of inpatient stay…
…The management of patients with hip fracture in whom a systolic murmur [indicating aortic stenosis (AS)] is heard remains conten- tious. Traditionally, anaesthetists have been reluctant to administer anaesthesia without additional echocardiographic information con- cerning aortic valve area, transvalvular gradient, and left ventricular contractility (indicated by ejection fraction), for fear of producing cerebral and coronary hypotension and ischaemia through arteriolar relaxation in patients with a relatively fixed, stenotic cardiac output, consequent to spinal (and general ) anaesthesia.
Guidelines have consistently stated that echocardiography is indicated if it has not been performed recently. However, despite the prevalence of AS being higher in the population with hip frac- ture (20–40% vs 3% in the over 75s, possibly contributing to the aetiology of the fall), several studies have found that early postoperative mortality among patients with AS undergoing hip fracture is similar to hip fracture patients without AS, although higher mortality has been noted in other studies. Insistence on pre- operative echocardiography has declined in recent years; however, as this can delay surgery, and the information yielded rarely changes management, which should be to treat patients with an audible ejection systolic murmur as if they had at least moderate AS, and administer anaesthesia accordingly, that is: using invasive arterial pressure monitoring and vasopressors to maintain coronary and cerebral perfusion pressure, and delivering anaesthesia sympa- thetically to the patients age and co-morbidities.
19 (Repeat) Electrocardiogram in the Cs5 configuration. What are you looking at when monitoring lead I.
A. anterior ischaemia B. atrial C. inferior D. lateral E. septal
D. Lateral
A. Aorta B. Hepatic artery C. Hepatic vein D. Portal pedicle E. Splenic Artery
D. Portal pedicle
both hepatic artery and portal vein
A. cease his omeprazole B. check his hepatic transaminase level C. check his renal function D. CHeck his QT interval on a resting ECG E. Decrease his oxycodone
C. Check his renal function
A. Coracobrachialis B. Deltiod C. Lat Dorsi D. Serratius Anterior E. Trapezius
C. Lat dorsi
A. Heat stress from anticholinergics B. Hypoxic ischaemic encephalopathy C. Neuroleptic malignant syndrome D. Serotonin syndrome E. Pain from fracture
C. Neuroleptic malignant syndrome
A) Well absorbed by Hb B) Poorly absorbed by H20 C) Widely disseminated in tissue D) Long infrared wavelength E) Short infrared wavelength
D. Long infrared wavelength