Modes of Ventilation - 26 Flashcards

1
Q

Describe VCV

A

TV selected

Constant flow rate until volume delivered

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2
Q

What effect does airflow at the end of inspiration during VCV have?

A

Peak Paw > Palveoli

The difference Paw - Palv = airway resistance

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3
Q

Advantages of VCV

A
  1. high airways resistance
  2. lung compliance dec

the ventilator still delivers desired volume

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4
Q

Disadvantages of VCV

A
  1. Higher airway pressures BUT they DO NOT increase risk of ventilator induced lung injury

Only an increase in alveolar pressure increases risk and this is the same in both VCV and PCV

  1. Constant inspiratory flow rate = short and uneven alveolar filling & inadequate with high flow demands –> decelerating pattern improves patient comfort
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5
Q

Describe PCV

A

Select inflation pressure + decelerating insp flow rate –> desired pressure quickly

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6
Q

Advantages of PCV

A
  1. Control peak alveolar pressure = reduce risk of injury

2. Better pt comfort - high initial flow rates, longer insp duration

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7
Q

At what pressure is ventilator-induced lung injury negligible?

A

Peak alveolar <30cmH2o

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8
Q

Disadvantages of PCV

A
  1. Dec alveolar volume when there is inc airway resistance or dec lung compliance = ARF
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9
Q

Describe PRVC

A

Constant TV but limits end-insp pressures

Measures lung compliance and selects lowest airway pressure needed to deliver desired TV

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10
Q

Describe Assist-Control Ventilation

A

Pt assisted or triggered BUT if not possible, vent triggered breaths @ pre-selected rate

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11
Q

Describes the vent triggers for ACV

A
  1. Patient triggered - pt generates -2 to -3 cm H2o –> opens pressure sens valve BUT this is double quiet breathing P = vent not always triggered
  2. Flow rate - little/no pressure change + flow 1-10L/min to trigger vent BUT system leaks –> auto-triggering
  3. Time-triggered - no neg pressure from pt = breath given by vent at preselected rate
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12
Q

Describe the goal of I:E

A

Usually 1:2

Allow enough exp time to prevent auto-PEEP

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13
Q

How do you increase I:E ratio if exhalation is too short?

A
  1. Inc insp flow rate
  2. Red TV
  3. Dec insp time (PCV)
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14
Q

Rapid breathing NOT d/t discomfort or anxiety - how do you dec resp rate? (pt unable to exhale completely)

A
  1. Sedation & insp flow adjustments = NOT successful

2. Use IMV

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15
Q

Describe IMV

A

Allows spontaneous breathing b/w vent breaths

Breathing circuit is parallel w/ vent w/ unidirectional valve that opens spontaneous breathing circuit when NO vent breath delivered.

SIMV is synchronized and can be volume or pressure-controlled

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16
Q

Adverse effects of IMV

A
  1. Inc work of breathing
  2. Dec CO - increases LV afterload

Both are d/t the spontaneous breathing period

17
Q

What accounts for the inc work of breathing during spontaneous breathing during IMV?

A

Resistance in the vent circuit –> use pressure support at 10cmH2o to reduce work of breathing

18
Q

Describe PSV

A

Pressure augmented spontaneous breathing - like PCV but allows patient to terminate inflation = pt control over insp time and TV

Lung inflation terminates when insp flow rate falls to 25% of peak flow rate = pt determines insp time and vol

19
Q

How can PSV be used to wean from the vent?

A

Using 5-10cmH2o to overcome resistance to flow from the vent tubing and airways = reducing work of breathing w/o augmenting TV

20
Q

Describe the pressure and collapse of airways during expiration

A

Progressive narrowing of distal airways during expiration –> collapse at end exp

Transpulmonary P where airways begin to collapse = closing pressure = 3cmH2o

Closing pressure HIGHER in small airway obstruction (COPD) & red lung compliance (ARDS)

21
Q

What are the adverse outcomes of airspace collapse at end-expiration?

A
  1. Impaired gas exchange from atelectasis

2. Atelectrauma from repetitive opening and closing of distal airspaced

22
Q

Describe the purpose of PEEP

A

To prevent airway pressure from < closing pressure at end expiration

23
Q

How does the vent create PEEP?

A

Pressure relief valve in exp limb –> exhalation proceeds until pressure falls to preselected level –> then mainteined

5-7cmH2o

24
Q

How does PEEP affect inflation pressures?

A

Upward

Higher Palv pressure and higher mean Pairway

  1. NOT related to PEEP level
  2. Change in Palv determines influence of PEEP on alv ventilation (art O2) and risk for volutrauma
25
Q

Describe the effects of different levels of PEEP

  1. 5-10 cmH2o
  2. 20-30 cmH2o
A
  1. prevents collapse

2. re-open distal airways persistently collapsed = alveolar recruitment = inc area for gas exchange

26
Q

What determines if PEEP will promote alveolar recruitment OR overdistension?

How do you determine which will occur?

A

Volume of recruitable lung (atelectasis that can be aerated, 2-25%)

  1. PEEP –> Lung compliance INC = recruitment (see ch. 25)
  2. PEEP –> PaO2/FiO2 INC = recruitment
27
Q

How does PEEP dec CO?

A
  1. Dec VR
  2. Dec vent compliance
  3. Inc RV afterload
  4. External constraint of ventricles

More prominent in hypovlemia

28
Q

How does PEEP affect O2 delivery?

A

Can inc alveolar recruitment and SaO2 BUT systemic O2 delivery may not improve d/t DEC CO

29
Q

At what point does one achieve optimal benefit from PEEP?

A

Greatest inc in systemic O2 delivery - measure at inc levels of PEEP