Peri-arrest arrhythmias Flashcards

1
Q

A-E assessment

A

Airway/breathing.

  • Assess airway and breathing status
  • Monitor SpO2 and RR
  • Give oxygen immediately to hypoxaemic patients (<94%) and adjust delivery according to oxygen saturations
  • ABG

Circulation.

  • Assess status (colour, CRT, pulses, BP, JVP, etc.)
  • Monitor HR and BP
  • Gain IV access
  • Venous gas: immediate Hb, U+Es, pH, glucose
  • Bloods sent to lab (FBC, U+Es, LFTs, clotting, calcium, CULTURES, glucose, ?troponins, ?group/save, ?TFTs)
  • IV fluids if indicated
  • ECG - assess rhythm, dictates treatment
  • Correct electrolyte abnormalities (eg. K+, Mg2+, Ca2+, glucose)
  • Note the presence or absence of ‘adverse features’
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2
Q

Assessing severity.

a) ‘Adverse features’ that indicate whether patient is stable or unstable (risk of death)
b) What must you check? - absence dictates switching to ALS algorithm

A

a) - Shock – hypotension (systolic BP <90), pallor, sweating, cold, clammy extremities, confusion or reduced GCS
- Syncope – transient LOC
- Myocardial ischaemia – typical ischaemic chest pain and/or typical ECG changes
- Heart failure – pulmonary oedema and/or raised JVP, +/- peripheral oedema and hepatomegaly

b) Pulse

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3
Q

4 basic management options for peri-arrest arrhythmias

- depends on nature of arrhythmia and whether patient is stable or unstable

A
  1. No treatment
  2. Simple manouevres (e.g. vagal stimulation)
  3. Drugs:
    - adrenaline
    - atropine
    amiodarone
  4. Electrical
    - cardioversion (tachy)
    - transcutaneous pacing (brady)
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4
Q

Tachyarrhythmia: management algorithm.

a) Initial management
b) Unstable (presence of ‘adverse features’)
c) If stable, must determine what 2 things to classify the rhythm?

A

a) - A-E
- Note presence of ‘adverse features’ (stable/unstable)
- Treat reversible causes (eg. electrolytes)

b) - Synchronised DC cardioversion up to 3 attempts*
- Then IV amiodarone 300 mg over 15 - 20 mins
- Repeat shock
- Then IV amiodarone 900 mg over 24 hours

  • Conscious patients require sedation or GA
  • synchronised DC is synchronised with the QRS, where defibrillation occurs randomly in the cardiac cycle

c) - Broad-complex (QRS > 120 ms) or narrow-complex?
- For each, is the rhythm regular or irregular?

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5
Q

Narrow-complex tachyarrhythmias: management*

a) If regular - 3 most likely causes and management
b) If irregular - likely cause and management

  • If stable - if presence of adverse features, revert to management of unstable tachyarrhythmia
A

a) Sinus tachycardia (NOT an arrhythmia):
- Treat underlying cause (eg. infection, blood loss, anaemia ,etc.)
- Do NOT give cardioversion or antiarrhythmic drugs

SVT (AVNRT):

  • Vagal manoeuvres
  • Adenosine - 6 mg rapid IV bolus; if no effect give 12 mg; if no effect again give further 12 mg
  • Monitor/record ECG continuously
Atrial flutter (2:1 AV block = HR ~ 150 bpm)
- Beta-blockade 

b) AF:
- If acute (< 48h) - consider electrical or chemical cardioversion
- Control rate with beta-blocker or diltiazem
- If in heart failure, consider digoxin or amiodarone
- Assess VTE risk and anticoagulate if necessary

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6
Q

Broad-complex tacharrhythmias: management*

a) If irregular - likely cause and management
b) If regular - 2 most likely causes and management

  • If stable - if presence of adverse features, revert to management of unstable tachyarrhythmia
A

a) AF with bundle branch block:
- Treat as per AF (BBs or CCB; digoxin or amiodaron in CCF)

b) - Monomorphic VT*: IV amiodarone 300 mg over 20 - 60 mins, then 900 mg over 24 hours
- SVT with bundle branch block: vagal manouevres, IV adenosine, beta-blockade, etc.

  • note: polymorphic VT (eg. Torsades) is highly likely to present with adverse features, requiring cardioversion
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7
Q

Torsades de Pointes.

a) Usual cause
b) ECG features
c) Management
d) Drug to avoid and why?

A

a) Ischaemia, QT prolongation (macrolides, TCAs, antipsychotics, hypoK+, hypoMg+, hypoCa2+, hypothermia, channelopathies)

b) - Broad-complex tachycardia
- Twisting of QRS complexes around the isoelectric line
- QT prolongation

c) - Stop all drugs known to prolong the QT interval
- Correct electrolyte abnormalities, especially low K+
- Give magnesium sulfate 2 g IV over 10 min (= 8 mmol, 4 mL of 50% magnesium sulfate)
- If adverse unstable (likely), immediate synchronised DC cardioversion under sedation or GA
- If the patient becomes pulseless, attempt defibrillation immediately (ALS algorithm)

d) Amiodarone - prolongs the QT (class III antiarrhythmic)

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8
Q

Administering adenosine.

A
  • Use a relatively large cannula and large vein (e.g. ACF)
  • Warn the patient that they will feel unwell and probably experience chest discomfort for a few seconds after the injection.
  • Initial: 6 mg as a rapid IV bolus.
  • Record an ECG during the injection.
  • If no response, give 12 mg, then again if necessary
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9
Q

AF with WPW.

a) Drugs to avoid and why?
b) Management

A

a) AV nodal blockers: adenosine, BBs, CCBs (diltiazem, verapamil) digoxin
b) Amiodarone

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10
Q

Bradycardia

a) Define bradycardia
b) Causes of bradycardia

A

a) HR < 60

b) - Physiological (e.g. during sleep, in athletes)
- Cardiac (e.g. AV block or SA node disease)
- Non-cardiac (e.g. vasovagal, hypothermia, hypothyroidism, hyperkalaemia)
- Drugs (e.g. beta-blockade, diltiazem, digoxin, amiodarone) in therapeutic use or overdose

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11
Q

Bradyarrhythmias.

a) Initial assessment
b) If any adverse features (unstable) - initial management
c) If stable, who to treat (risks of asystole)
d) Further management

A

a) A-E: monitor SpO2, oxygen if hypoxic, monitor HR and BP, ECG, IV access
- Look for any adverse features (shock, syncope, ischaemia, heart failure)
- Treat any reversible causes (eg. electrolytes, drugs)

b) IV atropine* 500 mcg
* Do not give in ischaemia (tachycardia may worsen infarct), or patients with heart transplant (denervated, may cause AV block or sinus arrest)

c) - Recent asystole, Mobitz type II / complete heart block, ventricular pause > 3 s
(if none present, continue observation)

d) - IV atropine 500 mcg can be given every 3 - 5 mins up to a maximum of 3 mg (6 doses)
- If these do not work, transcutaneous pacing is next best option
- If tc-Pacing not immediately available, consider other drugs (eg. isoprenaline, adrenaline, dopamine, glycopyrronium, theophylline)
- In extreme cases, may try fist pacing
- Transvenous pacing may be useful in Mobitz type II or complete heart block

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