What are classifications of wounds at surgery?
Clean: non-traumatic, uninfected operative wounds that only involve skin and musculoskeletal soft tissues
*Prophylactic abs in clean contaminated or worse
Clean-contaminated: Operative wounds where a hollow viscus (GIT/Resp/Urinary tract) is opened in a controlled manner with no spillage or contamination of surrounding tissues
Contaminated: During surgery bacteria has entered a normally sterile environement but for too brieg a period to allow infection to become established (leakage of intestinal contents into abdo cavity)
Dirty: Surgery is carried out to control an established infection (peritonitis/total ear canal abalation)
What are predictors of SSI?
What is the MIC breakpoint?
the chosen concentration of an antimicrobial which indicates whether a species of bacteria is susceptible or resistant to the antimicrobial
What are the 5 regions the abdomen can be divided into for exploration?
What are Halsted 6 surgical principles
What are the layers of the GIT
What are the stages of GIT healing
Lag phase: 1-3/4 days fibrin clot stage, minimal strength but will prevent some leakage - most likely stage to break down (72-96hrs)
Proliferation phase: 3/4-14 days fibroblasts causing epithelial migration and increased wound strength and immature collagen
Maturation phase: 14-180 days, less clinical importance reorganisation and remodelling of collagen
What can the omentum be used for?
helps to seal leaks
helps bring nutrients and lymphatic drainage
sutures v. wrapping
What is the use of serosal patching?
How to carry out an enterotomy
Closing an enterectomy
Disparity of the lumens:
- spatulating smaller lumen at antimesenteric border
- larger lumen be partially closed with a simple continuous pattern until to matches that of remaining colon or proximal duodenum
What are alternatives to suturing GIT
What are causes of small bowel obstruction
Intraluminal, Intramural, Extramural
1. FB
2. Tumors
3. Strictures due to trauma/prior surgery
4. Intussusception
5. Strangulation
6. Abscesses
7. Adhesions
Pathophysiology of invagination and causes
Invagination of a portion of intestine due to vigorous contraction of one segment into a relaxed segment.
Causes: young dogs: parasitism due to enteritis, older dog: neoplasia
Blood supply to intussuscepted portion of intestine is compromised due to inclusion in invagination. Initially venous occlusion with edema of the bowek and if prolonged arterial occlusion and necrosis
Eventually fibrous adhesions can form making spontaneous or surgical reduction difficult
following surgery can recurr up to 20% with 72hrs of procedure
Prognostic factors useful for GDV
GDV Patient Stabilisation
Newer options/minimal published data
1. Corticosteroids: no published evidence - perceived effects antioxidant effect, increased vascular tone and positive inotropic effect
2. Deferoxamine and allopurinol - free radical scavengers
Paracetamol after stable and post surgery
No NSAIDs
Options for gastric decompression
What are the goals of GDV surgery?
What is the surgical approach to a GDV?
How do you assess gastric/splenic viability?
Color - black/blue/green/black
Touch - friability
Pulses
Bleeding
The greater curvature and fundus are most likely to be ischaemic
Gastrectomy without stapling device - 60% mortality
Invagination is an option - meleana to be expected
GDV post op care
Types of peritonitis
Primary peritonitis - no obvious source of contamination, haematogenous spread of bacteria - FIP
Secondary peritonitis:
- Septic (GI rupture/perforation/dehisence, penetrating trauma, blunt trauma, AI drugs, FB rupture)
- Aseptic: sterile mechanial or chemical irritation of the peritoneal cavity (pancreatic enzymes, gastric enzymes, talcum powder from gloves, barium, urine/bile, fb)
localised/generalised peritonitis