1
Q
Latin: “Mors”
A
Irreversible cessation of life of the whole organism
2
Q
Death can be either…
A
- Natural
- Pathological
3
Q
Atria mortis
A
Death caused by a defect of either:
- Brain
- Heart
- Lungs
4
Q
Sudden death
A
Mors subita
- Loss of senses
- Constrictions
- Convulsions
- Final relaxation of muscles
5
Q
Clinical death
A
- Resp/HR Ø
- Tissues and organs survive for a while
6
Q
Give the course of clinical death
A
- Clinical death
- Pathological agony
- Pathological/absolute death
7
Q
Recognition of death
A
- No heartbeat
- No response to sensory or sensitive stimuli
- No reflexes (Pupil etc.
- Post mortem changes
Pathology A - General Pathology
flashcards
Decks in class (85)
# Cards
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[A] 1.15 Spread of the pathological processes in the organism. Septicaemia, sepsis, bacteriaemia33
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[A] 1.17 Local hyperaemia25
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[A] 1.18 Local oligaemia (ischaemia)23
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[A] 1.19 Hemostasis, shock30
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[A] 1.20 Haemorrhage24
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[A] 1.21 Thrombosis22
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[A] 1.22 Embolism11
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[A] 1.38 Necrosis & Apoptosis36
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[A] 1.41 About the regeneration in general, removal of the necrotic tissues11
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[A] 1.43 Organisation, demarcation11
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[A] 1.44 Regeneration of epithelial tissue11
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[A] 1.45 Regeneration of muscle and neural tissue6
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[A] 1.46 Regeneration of connective tissue and blood vessels. Wound healing26
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[A] 1.50 Vascular changes and exudation in the acute inflammation31
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[A] 1.52 Proliferative processes during inflammation.7
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[A] 1.51 Infiltrative processes during acute inflammation22
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[A] 1.59 Tuberculum. Predominantly exudative and predominantly proliferative tuberculosis29
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[A] 1.53 Serous, catarrhal, haemorrhagic and ichorous inflammation25
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[A] 1.54 Suppurative inflammation16
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[A] 1.55 Fibrinous inflammation18
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[A] 1.60 Factors influencing the character of tuberculosis11
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[A] 1.61 General pathogenesis of tuberculosis37
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[A] 1.70 The general macro- and microscopic morphology of tumors. The increase in volume of tumor cells and tumoral proliferations33
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[A] 1.71 Tumor-induced angiogenesis (capillary incorporation, budding of endothelial cells)8
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[A] 1.74 The tumor-organism relationship (tumor progression, heterogeneity, metabolism of tumor cells, cancer anorexia-cachexia syndrome, paraneoplasia, tumor immunity)23
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[A] 1.75 Metastasis24
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[A] 1.72 Carcinogenesis (chemical and physical agents)25
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[A] 1.73 Carcinogenesis (biological agents)9
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[A] 1.83 Rules of collecting and sending specimens for pathological and for other supplementary examinations5
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[B] 1.6 High temperature as cause of disease23
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[B] 1.7 Low temperature as a cause of disease8
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[B] 1.9 Inadequate oxygen and water supply12
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[B] 1.10 Inadequate nutrient supply. Starvation.9
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[B] 1.11 Autointoxication46
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[B] 1.13 Infectious agents/Bacteria, viruses and fungi as causes of disease22
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[B] 1.23 Disturbances in water balance of the organism (Edema)18
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[B] 1.14 Pathological aspects of immunology23
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[B] 1.24 Disturbances in water balance of cells7
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[B] 1.25 Disturbances in fat metabolism19
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[B] 1.26 Disturbances in carbohydrate metabolism15
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[B] 1.28 Disturbances in keratinization21
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[B] 1.29 Uricosis (Gout)14
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[B] 1.31 Amyloidosis21
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[B] 1.40 Pathological calcification10
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[B] 1.48 Atrophy13
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[B] 1.62 Actinobacillosis, actinomycosis28
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[B] 1.65 Disturbances in cell division25
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[B] 1.68 Definition of tumor (oncogenes, tumorsuppressor genes)28
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[B] 1.69 Terminology and classification of tumors (beningn, semimalignant tumors, in situ carcinoma, malignant tumors, preneoplastic changes, tumor-like lesions)29
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[B] 1.76 Benign mesenchymal tumors13
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[B] 1.77 Malignant mesenchymal tumors4
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[B] 1.78 Melanocytic tumors, the important tumors of nervous tissue. Mixed tumors18
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[B] 1.79 Benign epithelial tumors8
-
[B] 1.80 Malignant epithelial tumors15
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[B] 1.81 Tumors of vascular tissues (blood and lymph vessels)9
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[B] 1.82 Hemolymphatic tumors in mammals21
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[B] 1.84 Basic principles and technique of histopathological examinations. Light microscopy.2
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[C] 1.5 Traumas and their consequences11
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[C] 1.1 Objective, task and investigation methods of pathology19
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[C] 1.2 Disease13
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[C] 1.3 Death7
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[C] 1.4 Postmortem changes32
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[C] 1.8 Irradiations and electricity as cause of disease32
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[C] 1.12 Internal Conditions of development of diseases15
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[C] 1.16 Degenerative changes of cells and tissues21
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[C] 1.27 Pathological mucus production and pathological changes in the mucosubstance11
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[C] 1.30 Hyalinosis, fibrinoid necrosis21
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[C] 1.32 Pathologic pigmentation caused by hemoglobin and haemosiderin6
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[C] 1.33 Pathologic pigmentation caused by hematoidin, and bilirubin7
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[C] 1.34. Pathologic pigmentation caused by porphyrins6
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[C] 1.35 Pathologic pigmentation caused by melanin, lipofuscin, and ceroid25
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[C] 1.36 Exogenous pigments20
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[C] 1.37 Concretions (lithiasis) and pseudoconcretions24
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[C] 1.39 Gangrene8
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[C] 1.42 Proliferative changes during regeneration. Fibrosis, elastosis, fibroelastosis, reticulosis, cirrhosis27
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[C] 1.47 Metaplasia, pseudomateplasia9
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[C] 1.49 Accomodation (hypertrophy, hyperplasia, transformatio)17
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[C] 1.56 Inflammation in organs without blood vessels. The inflammation-organism relationship27
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[C] 1.57 Necrotizing and proliferative inflammation7
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[C] 1.58 General characteristics of the granuloma formation20
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[C] 1.63 Granulomas caused by fungi28
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[C] 1.64 Granulomas caused by chemicals and other granulomas10
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[C] 1.66 Causes of developmental anomalies (formal and causal genesis)13
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[C] 1.67 Main forms of developmental anomalies28
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[C] 1.85 Professional and formal aspects of writing a post mortem report5
